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Improving pharmacogenetic prediction of extrapyramidal symptoms induced by antipsychotics

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Thank you for visiting nature. You are using a browser version with limited support for CSS. To obtain the best experience, we recommend you use a more up to date browser or turn off compatibility mode in Internet Explorer. In the meantime, to ensure continued support, we are displaying the site without styles and JavaScript. In previous work we developed a pharmacogenetic predictor of antipsychotic AP induced extrapyramidal symptoms EPS based on four genes involved in mTOR regulation.

The main objective is to improve this predictor by increasing its biological plausibility and replication. We re-sequence the four genes using next-generation sequencing. The result is an improved pharmacogenetic predictor of AP-induced EPS, which is more robust and generalizable than the original. Antipsychotic AP medication is the gold standard in schizophrenia treatment.

Although APs have demonstrated overall efficacy and safety there are large inter-individual differences in their efficacy and side effects between patients. One of the strongest predictors of noncompliance is the experience of harmful side effects 2.

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